Consolidating new memories requires the amygdala and

The result of the gene expression is the lasting alteration of synaptic proteins, as well as synaptic remodeling and growth.

LTP, one of the best understood forms of synaptic plasticity, is thought to be a possible underlying process in synaptic consolidation.

The case of Henry Molaison, formerly known as patient H.

M., became a landmark in studies of memory as it relates to amnesia and the removal of the hippocampal zone and sparked massive interest in the study of brain lesions and their effect on memory.

and occurs within the first few hours after learning, and systems consolidation, where hippocampus-dependent memories become independent of the hippocampus over a period of weeks to years.

Recently, a third process has become the focus of research, reconsolidation, in which previously-consolidated memories can be made labile again through reactivation of the memory trace.Specifically, NMDA-receptor antagonists appear to block the induction of both LTP and fear conditioning and that fear conditioning increases amygdaloidal synaptic transmission that would result in LTP.

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